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<math>
\alpha + 444444 + \beta + \omega
</math>
jepjepjepjep
[[:File:aditro.PNG]]
[[Image:kuvatiedostonmitähäan.jpg]]
<display_map type="terrain">
62.904602, 27.647781
62.891918, 27.680013
</display_map>
{{mfiles}}
<t2b index="Make" unit="kg" desc="Description">
Lata|100|
Toyota|88|Toijota
</t2b>
{{variable|moderator=Ehac}}
{{variable|moderator=Ehac}}
mitä tapahtuu!


http://ytoswww/yhteiset/YMAL/Projects/


Edited by ehac
 
<math>\alpha + 8884444</math>


<math>
<math>
\textstyle


L_{Aeq} = 10 \log \int_{t_0}^{t_1} \frac{p_A^2(t)}{p_0^2} dt
L_{Aeq} = 10 \log \int_{t_0}^{t_1} \frac{p_A^2(t)}{p_0^2} dt
Line 11: Line 37:
</math>
</math>


== Live Code Test ==
<rcode live=1 graphics=1 name='live_test' variables='name:breaks|description:Breaks|options:10;10;20;20;35;35;50;50|type:selection|default:10'>
hist(faithful$eruptions,
      probability = TRUE,
      breaks = as.numeric(breaks),
      xlab = "Duration (minutes)",
      main = "Geyser eruption duration")
</rcode>
== Sotkanet ==
<rcode name='sotkanet_test'>
library(OpasnetUtils)
library(OpasnetUtilsExt)
# collect makes a data.frame out of the list object from Sotkanet
# x is the input data
# name is the name for the column
# single is a logical whether there is only a single entry in the x data.
collect <- function(x, name, single = FALSE) {
out <- data.frame()
if(single) {out <- data.frame(temp1 = x$id, temp2 = x$title$fi)
} else {
for(i in 1:length(x)) {
out <- rbind(out, data.frame(temp1 = x[[i]]$id, temp2 = x[[i]]$title$fi))
}
}
colnames(out) <- c(name, paste(name, "Result", sep=""))
return(out)
}
a <- sotkanet.indicators()
# print(a)
b <- sotkanet.indicators(127)
b <- collect(b, "indicator", TRUE)
# print(xtable(b), type = 'html')
d <- sotkanet.regions()
d <- collect(d, "region")
# print(xtable(d), type = 'html')
e <- sotkanet.data(indicator=127,years=c(2011,2010),genders='female')
e <- merge(b, e)
e <- merge(d, e)
oprint(e)
</rcode>
== Objects save test ==
<rcode name='objs_save_test'>
library(OpasnetUtils)
x <- stats::runif(20)
y <- list(a = 1, b = TRUE, c = "Jeah baby jeah!")
objects.store(x, y, verbose=TRUE)
</rcode>
==Giving tables via user interface==
<rcode embed="1" variables="name:test|type:table">
library(OpasnetUtils)
oprint(test)
</rcode>
== ovariable merge testing ==
<rcode>
library(OpasnetUtils)
aa <- new("ovariable", output = data.frame(dummy=NA))
bb <- new("ovariable", output = data.frame(a=1:4))
#cc <- new("ovariable", output = data.frame(a=1:4))
cc <- ''
test <- Ovariable(
name='test',
dependencies = data.frame(
Name = c("aa", "bb", "cc"),
Ident = c(NA, NA, NA)
),
formula = function(dependencies, ...) {
ComputeDependencies(dependencies, ...)
out <- merge(aa, bb)
return(out)
}
)


<math>
oprint(test)
\textstyle


L_{Aeq} = 10 \log \int_{t_0}^{t_1} \frac{p_A^2(t)}{p_0^2} dt


</math>
</rcode>


== Static GoogleMaps test ==
== Static GoogleMaps test ==
Line 28: Line 152:
library(RColorBrewer)
library(RColorBrewer)
library(classInt)
library(classInt)
library(OpasnetBaseUtils)
library(OpasnetUtils)
library(OpasnetUtilsExt)


shp<-readOGR('PG:host=localhost user=postgres dbname=spatial_db','kuopio_house')
shp<-readOGR('PG:host=localhost user=postgres dbname=spatial_db','kuopio_house')
Line 97: Line 222:
legend("topleft", legend=names(attr(colcode, "table")),title="Ika", fill=attr(colcode, "palette"),  cex=1.0, bty="y",bg="white")
legend("topleft", legend=names(attr(colcode, "table")),title="Ika", fill=attr(colcode, "palette"),  cex=1.0, bty="y",bg="white")
</rcode>
</rcode>


== Kuopio buildings on Google maps test ==
== Kuopio buildings on Google maps test ==
Line 108: Line 231:
library(RColorBrewer)
library(RColorBrewer)
library(classInt)
library(classInt)
library(OpasnetBaseUtils)
library(OpasnetUtils)
library(OpasnetUtilsExt)
library(RODBC)
library(RODBC)


Line 185: Line 309:
</rcode>
</rcode>


 
== GoogleMaps Sorvi MML TEST ==
=== GoogleMaps Sorvi MML TEST ===


<rcode name='gmapspsqltest3'>
<rcode name='gmapspsqltest3'>


library(OpasnetBaseUtils)
library(OpasnetUtils)
library(OpasnetUtilsExt)
library(sorvi)
library(sorvi)
library(rgdal)
library(rgdal)
Line 217: Line 341:
</rcode>
</rcode>


=== GoogleMaps PostgreSQL test 2 ===
== GoogleMaps PostgreSQL test 2 ==


<rcode name='gmapspsqltest2'>
<rcode name='gmapspsqltest2'>
Line 225: Line 349:
library(RColorBrewer)
library(RColorBrewer)
library(classInt)
library(classInt)
library(OpasnetBaseUtils)
library(OpasnetUtils)
library(OpasnetUtilsExt)


shp<-readOGR('PG:host=localhost user=postgres dbname=spatial_db','watson_wkt')
shp<-readOGR('PG:host=localhost user=postgres dbname=spatial_db','watson_wkt')
Line 252: Line 377:
</rcode>
</rcode>


=== GoogleMaps PostgreSQL test ===
== GoogleMaps PostgreSQL test ==


<rcode name='gmapspsqltest'>
<rcode name='gmapspsqltest'>
library('OpasnetBaseUtils')
library(OpasnetUtils)
cat("<span style='font-size: 1.2em;font-weight:bold;'>PostgreSQL Test</span>\n")
library(OpasnetUtilsExt)
cat("PostgreSQL Test\n")
google.show_data_on_maps()
google.show_data_on_maps()


Line 264: Line 390:
</rcode>
</rcode>


=== Opasnet.csv test ===
== Opasnet.csv test ==


<rcode name='opcsvtest'>
<rcode name='opcsvtest'>
library(OpasnetBaseUtils)
library(OpasnetUtils)


csv <- opasnet.csv("2/25/Russian_elections_2011_results.csv")
csv <- opasnet.csv("2/25/Russian_elections_2011_results.csv")
Line 275: Line 401:
</rcode>
</rcode>


=== Opasnet.data and BUGS test ===
== Opasnet.data and BUGS test ==


<rcode name='odabugstest' graphics=1>
<rcode name='odabugstest' graphics=1>
library(OpasnetBaseUtils)
library(OpasnetUtils)
pumps.model <- opasnet.data('c/cc/Test_bugs_model.txt')
pumps.model <- opasnet.data('c/cc/Test_bugs_model.txt')


Line 329: Line 455:


<br> {{greenbox|
<br> {{greenbox|
===Hello===
==Hello==
* this works
* this works
}}  
}}  


{{bluebox|
{{bluebox|
===Bluebox===
==Bluebox==
* works
* works
* ok
* ok
Line 353: Line 479:
name:divisions2|description:Minkä yhden tekijän halua eritellä kuvaajassa?|type:selection|options:'Katalyytin määrä';Katalyytin määrä;'Ikä';Ikä;'Kastelu';Kastelu;'Käytetty puristin';Käytetty puristin|default:'Kastelu'
name:divisions2|description:Minkä yhden tekijän halua eritellä kuvaajassa?|type:selection|options:'Katalyytin määrä';Katalyytin määrä;'Ikä';Ikä;'Kastelu';Kastelu;'Käytetty puristin';Käytetty puristin|default:'Kastelu'
">
">
library(OpasnetBaseUtils)
library(OpasnetUtils)
library(ggplot2)
library(ggplot2)
library(xtable)
library(xtable)
Line 441: Line 567:
</rcode>
</rcode>


&nbsp;<!-- __OBI_TS:1332833567 -->
=== MassHEIS test ===


==Kristiina telmii==
:''Moved to [[MassHEIS]].


<t2b index = "Substance,Observation type" locations = "CASRN,WOE 86 Guidelines,WOE Narrative,IRIS identifier" unit = "no unit">
=== MassHEIS test multilayer NOT WORKING YET ===
Acenaphthene|83-32-9|Not Assessed under the IRIS program.||0442
<rcode name='MassHEISTestML' graphics='1'>
Acenaphthylene| 208-96-8|D, Not classifiable as to human carcinogenicity|Based on no human data and inadequate data from animal bioassays.|0443
library(OpasnetUtils)
Acephate|30560-19-1|C, Possible human carcinogen|The classification is based on increased incidence of hepatocellular carcinomas and adenomas in female mice.|0354
library(OpasnetUtilsExt)
Acetaldehyde|75-07-0|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on increased incidence of nasal tumors in male and female rats and laryngeal tumors in male and female hamsters after inhalation exposure.|0290
library(ggplot2)
Acetochlor|34256-82-1|Not Assessed under the IRIS program.||0521
library(rgdal)
Acetone|67-64-1|NA, Not applicable. This substance was not assessed using the 1986 cancer guidelines (U.S. EPA, 1986).|In accordance with the Draft Revised Guidelines for Carcinogen Risk Assessment (U.S. EPA, 1999) data are inadequate for an assessment of the human carcinogenic potential of acetone.|0128
library(maptools)
Acetonitrile|75-05-8|D, Not classifiable as to human carcinogenicity|Under the Proposed Guidelines for Carcinogen Risk Assessment (U.S. EPA, 1996), the carcinogenic potential of ACN following inhalation, oral, or dermal exposure is best characterized as "cannot be determined because the existing evidence is composed of conflicting data (e.g., some evidence is suggestive of carcinogenic effects, but other equally pertinent evidence does not confirm any concern)."|0205
library(RColorBrewer)
Acetophenone|98-86-2|D, Not classifiable as to human carcinogenicity|Based on no human data and no animal data.|0321
library(classInt)
Acetyl chloride|75-36-5|D, Not classifiable as to human carcinogenicity|No human data or animal data.|0518
library(raster)
Acifluorfen, sodium|62476-59-9|Not Assessed under the IRIS program.||0192
 
Acrolein|107-02-8|NA, Not applicable. This substance was not assessed using the 1986 cancer guidelines (U.S. EPA, 1986).|Under the Draft Revised Guidelines for Carcinogen Risk Assessment (U.S. EPA, 1999), the potential carcinogenicity of acrolein cannot be determined because the existing data are inadequate for an assessment of human carcinogenic potential for either the oral or inhalation route of exposure. There are no adequate human studies of the carcinogenic potential of acrolein. Collectively, experimental studies provide inadequate evidence that acrolein causes cancer in laboratory animals.|0364
data <- MassHEIS.data()
Acrylamide|79-06-1|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on inadequate human data and sufficient evidence of carcinogenicity in animals; significantly increased incidences of benign and/or malignant tumors at multiple sites in both sexes of rats, and carcinogenic effects in a series of one-year limited bioassays in mice by several routes of exposures. The classification is supported by positive genotoxicity data, adduct formation activity, and structure-activity relationships to vinyl carbamate and acrylonitrile.|0286
 
Acrylic acid|79-10-7|Not Assessed under the IRIS program.||0002
# Plot the data
Acrylonitrile|107-13-1|B1, Probable human carcinogen - based on limited evidence of carcinogenicity in humans|The observation of a statistically significant increase in incidence of lung cancer in exposed workers and observation of tumors, generally astrocytomas in the brain, in studies in two rat strains exposed by various routes (drinking water, gavage, and inhalation) forms the basis for this classification.|0206
coordinates(data)=c("longitude","latitude")
Adiponitrile|111-69-3|D, Not classifiable as to human carcinogenicity|No human and no animal cancer data were available. Adiponitrile was negative for mutagenicity in Salmonella with and without activation.|0515
proj4string(data)<-("+init=epsg:4326")
Alachlor|15972-60-8|Not Assessed under the IRIS program.||0129
epsg4326String <- CRS("+proj=longlat +ellps=WGS84 +datum=WGS84 +no_defs")
Alar|1596-84-5|Not Assessed under the IRIS program.||0287
shp<-spTransform(data,epsg4326String)
Aldicarb|116-06-3|D, Not classifiable as to human carcinogenicity|Aldicarb was not found to induce statistically significant increases in tumor incidence in mice or rats in feeding studies or mice in a skin painting study. In the feeding studies there were, however, significant trends in pituitary tumors in female rats and fibrosarcomas in the male mouse. This evidence, together with the fact that less than maximum tolerated doses were used, indicates that the available assays are inadequate to assess the carcinogenic potential of aldicarb.|0003
 
Aldicarb sulfone|1646-88-4|Not Assessed under the IRIS program.||0312
 
Aldrin|309-00-2|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Orally administered aldrin produced significant increases in tumor responses in three different strains of mice in both males and females. Tumor induction has been observed for structurally related chemicals, including dieldrin, a metabolite.|0130
start <- min(shp$anmean)
Ally|74223-64-6|Not Assessed under the IRIS program.||0288
end <- max(shp$anmean)
Allyl alcohol|107-18-6|Not Assessed under the IRIS program.||0004
 
Allyl chloride|107-05-1|C, Possible human carcinogen|Classification is based on a low (but biologically important) incidence of forestomach tumors in female mice and positive results in a variety of genetic toxicity tests. Allyl chloride is an alkylating agent and structurally related to probable human carcinogens.|0387
rasters = list()
Aluminum phosphide|20859-73-8|Not Assessed under the IRIS program.||0005
years = c(2000,2001,2002)
Amdro|67485-29-4|Not Assessed under the IRIS program.||0207
 
Ametryn|834-12-8|Not Assessed under the IRIS program.||0208
for(y in 1:length(years))
4-Aminopyridine|504-24-5|D, Not classifiable as to human carcinogenicity|No human data and no animal data available.|0440
{
Amitraz|33089-61-1|Not Assessed under the IRIS program.||0334
#Create blank raster
Ammonia|7664-41-7|Not Assessed under the IRIS program.||0422
rast<-raster()
Ammonium acetate|631-61-8|D, Not classifiable as to human carcinogenicity|No human data and no animal data|0517
 
Ammonium methacrylate|16325-47-6|D, Not classifiable as to human carcinogenicity|No human data and no animal data|0516
s <- shp[(shp$Year == years[y]),]
Ammonium sulfamate|7773-06-0|Not Assessed under the IRIS program.||0007
 
Aniline|62-53-3|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Induction of tumors of the spleen and the body cavity in two strains of rat, and some supporting genetic toxicological evidence.|0350
#Set raster extent to that of point data
ortho-Anisidine|90-04-0|Not Assessed under the IRIS program.||0610
extent(rast)<-extent(s)
Anthracene|120-12-7|D, Not classifiable as to human carcinogenicity|Based on no human data and inadequate data from animal bioassays.|0434
 
Antimony|7440-36-0|Not Assessed under the IRIS program.||0006
#Choose number of columns and rows
Antimony trioxide|1309-64-4|Not Assessed under the IRIS program.||0676
ncol(rast) <- 64
Apollo|74115-24-5|C, Possible human carcinogen|Based on an increase in thyroid gland follicular cell tumors in male rats and supportive findings in pituitary/thyroid hormone activity.|0008
nrow(rast) <- 64
Aramite|140-57-8|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on no human data and sufficient data from animal bioassays including increased incidence of liver tumors and/or neoplastic nodules in three strains of male and female rats and males of one strain of mice, and extrahepatic biliary system tumors in dogs following chronic oral exposure.|0473
 
Aroclor 1016|12674-11-2|Not Assessed under the IRIS program.||0462
#Rasterize point data
Aroclor 1248|12672-29-6|Not Assessed under the IRIS program.||0649
rasters[[y]] <- rasterize(s, rast, s$anmean, fun=mean)
Aroclor 1254|11097-69-1|Not Assessed under the IRIS program.||0389
}
Arsenic, inorganic|7440-38-2|A, Human Carcinogen| Based on sufficient evidence from human data. An increased lung cancer mortality was observed in multiple human populations exposed primarily through inhalation. Also, increased mortality from multiple internal organ cancers (liver, kidney, lung, and bladder) and an increased incidence of skin cancer were observed in populations consuming drinking water high in inorganic arsenic.|0278
 
Arsine|7784-42-1|Not Assessed under the IRIS program.||0672
steps <- approx(c(start,end),n=6)$y
Asbestos|1332-21-4|A, Human Carcinogen|Observation of increased mortality and incidence of lung cancer, mesotheliomas and gastrointestinal cancer in occupationally exposed workers are consistent across investigators and study populations. Animal studies by inhalation in two strains of rats showed similar findings for lung cancer and mesotheliomas. Animal evidence for carcinogenicity via ingestion is limited (male rats fed intermediate-range chrysotile fibers; i.e., >10 um length, developed benign polyps), and epidemiologic data in this regard are inadequate.|0371
colors <- rev(rainbow(length(steps), start=0, end=0.50))
Assure|76578-14-8|D, Not classifiable as to human carcinogenicity|Based on no human data and inadequate animal data.|0335
 
Asulam|3337-71-1|Not Assessed under the IRIS program.||0284
par(mfrow=c(6,1), mar=c(3,1,0,1), cex=1.5)
Atrazine|1912-24-9|Not Assessed under the IRIS program.||0209
 
Avermectin B1|65195-55-3|Not Assessed under the IRIS program.||0381
colorstrip <- function(colors, labels)
Azobenzene|103-33-3|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Azobenzene induced invasive sarcomas in the spleen and other abdominal organs in male and female F344 rats following dietary administration. It is genotoxic and may be converted to benzidine, a known human carcinogen, under the acidic conditions in the stomach.|0351
{
Barium and Compounds|7440-39-3|D, Not classifiable as to human carcinogenicity|Under the Proposed Guidelines for Carcinogenic Risk Assessment (U.S. EPA, 1996), barium is considered not likely to be carcinogenic to humans following oral exposure and its carcinogenic potential cannot be determined following inhalation exposure.|0010
count <- length(colors)
Barium cyanide|542-62-1|Not Assessed under the IRIS program.||0009
m <- matrix(1:count, count, 1)
Baygon|114-26-1|Not Assessed under the IRIS program.||0210
image(m, col=colors, ylab="", axes=FALSE)
Bayleton|43121-43-3|Not Assessed under the IRIS program.||0131
axis(1,approx(c(0, 1), n=length(labels))$y, labels)
Baythroid|68359-37-5|Not Assessed under the IRIS program.||0132
}
Benefin|1861-40-1|Not Assessed under the IRIS program.||0133
 
Benomyl|17804-35-2|Not Assessed under the IRIS program.||0011
cat("<span style='font-size: 1.2em;font-weight:bold;'>Massachusetts annual mean PM 2.5 microns</span>\n")
Bentazon (Basagran)|25057-89-0|E, Evidence of non-carcinogenicity for humans|Under EPA's proposed guidelines for carcinogen risk assessment (U.S. EPA, 1996), Bentazon would be characterized as not likely to be carcinogenic to humans by any route of exposure.|0134
 
Benz[a]anthracene|56-55-3|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on no human data and sufficient data from animal bioassays. Benz[a]anthracene produced tumors in mice exposed by gavage; intraperitoneal, subcutaneous or intramuscular injection; and topical application. Benz[a]anthracene produced mutations in bacteria and in mammalian cells, and transformed mammalian cells in culture.|0454
colorstrip(colors, steps)
Benzaldehyde|100-52-7|Not Assessed under the IRIS program.||0332
 
Benzene|71-43-2|A, Human Carcinogen|Under the proposed revised Carcinogen Risk Assessment Guidelines (U.S. EPA, 1996), benzene is characterized as a known human carcinogen for all routes of exposure based upon convincing human evidence as well as supporting evidence from animal studies. (U.S. EPA, 1979, 1985, 1998; ATSDR, 1997).|0276
#Plot data
Benzidine|92-87-5|A, Human Carcinogen|Observation of increased incidence of bladder cancer and bladder cancer-related deaths in exposed workers|0135
 
Benzo[a]pyrene (BaP)|50-32-8|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Human data specifically linking benzo[a]pyrene (BAP) to a carcinogenic effect are lacking. There are, however, multiple animal studies in many species demonstrating BAP to be carcinogenic following administration by numerous routes. BAP has produced positive results in numerous genotoxicity assays.|0136
s <- stack(rasters)
Benzo[b]fluoranthene|205-99-2|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on no human data and sufficient data from animal bioassays. Benzo[b]fluoranthene produced tumors in mice after lung implantation, intraperitoneal (i.p.) or subcutaneous (s.c.) injection, and skin painting.|0453
names(s) <- c('z2000','z2001','z2002')
Benzo[g,h,i]perylene|191-24-2|D, Not classifiable as to human carcinogenicity|Based on no human data and inadequate animal data from lung implant, skin-painting and subcutaneous injection bioassays.|0461
 
Benzo[k]fluoranthene|207-08-9|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on no human data and sufficient data from animal bioassays. Benzo[k]fluoranthene produced tumors after lung implantation in mice and when administered with a promoting agent in skin-painting studies. Equivocal results have been found in a lung adenoma assay in mice. Benzo[k]fluoranthene is mutagenic in bacteria.|0452
print(s)
Benzoic acid|65-85-0|D, Not classifiable as to human carcinogenicity|No human data and inadequate data from animal bioassays|0355
 
Benzotrichloride|98-07-7|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on inadequate human data and sufficient evidence of carcinogenicity in animals; namely, significantly increased incidences of benign and malignant tumors at multiple sites in one strain of female mice treated orally, dermally, and by inhalation. There is also evidence of mutagenicity in a variety of test systems.|0388
Benzyl chloride|100-44-7|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on inadequate human data and sufficient evidence of carcinogenicity in animals; namely significantly increased incidences of benign and malignant tumors at multiple sites in both sexes of mice and a significant increase in thyroid tumors in female rats. There was evidence of mutagenicity in a variety of test systems.|0393
 
Beryllium and compounds|7440-41-7|B1, Probable human carcinogen - based on limited evidence of carcinogenicity in humans|Using the 1996 proposed Guidelines for Carcinogen Risk Assessment, inhaled beryllium would be characterized as a "likely" carcinogen in humans, and the human carcinogenic potential of ingested beryllium cannot be determined.|0012
google.show_raster_on_maps(s, col=colors, style="height:500px;")
Bidrin|141-66-2|Not Assessed under the IRIS program.||0211
 
Biphenthrin|82657-04-3|Not Assessed under the IRIS program.||0333
 
1,1-Biphenyl|92-52-4|D, Not classifiable as to human carcinogenicity|No human data and inadequate studies in mice and rats. Results of genotoxicity tests are generally negative.|0013
</rcode>
Bis(2-chloro-1-methylethyl) ether|108-60-1|Not Assessed under the IRIS program.||0407
 
Bis(2-chloroethoxy)methane|111-91-1|D, Not classifiable as to human carcinogenicity|No human data and no animal data.|0522
 
Bis(chloroethyl)ether (BCEE)|111-44-4|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Positive carcinogenicity results in two strains of mice and evidence of mutagenicity|0137
 
Bis(chloromethyl)ether (BCME)|542-88-1|A, Human Carcinogen|Statistically significant increases in lung tumors (oat cell carcinomas) observed in six studies of exposed workers and bioassay data from rats and mice.|0375
&nbsp;<!-- __OBI_TS:1353932360 -->
Bisphenol A.|80-05-7|Not Assessed under the IRIS program.||0356
Boron and Compounds|7440-42-8|||0410
Bromate|15541-45-4|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Under the Proposed Guidelines for Carcinogen Risk Assessment (U.S. EPA, 1996), Bromate should be evaluated as a likely human carcinogen by the oral route of exposure. Insufficient data are available to evaluate the human carcinogenic potential of Bromate by the inhalation route.|1002
Brominated dibenzofurans|NA|D, Not classifiable as to human carcinogenicity|No data in humans or animals|0514
Bromobenzene|108-86-1|||1020
Bromochloromethane|74-97-5|D, Not classifiable as to human carcinogenicity|Based on the lack of data regarding the carcinogenicity of bromochloromethane in humans or animals; however, there are data indicative of genotoxic effects and structural relationships to halogenated methanes classified as B2 probable human carcinogens.|0532
Bromodichloromethane|75-27-4|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on inadequate human data and sufficient evidence of carcinogenicity in two animal species (mice and rats) as shown by increased incidence of kidney tumors and tumors of the large intestine in male and female rats, kidney tumors in male mice, and liver tumors in female mice.|0213
p-Bromodiphenyl ether|101-55-3|D, Not classifiable as to human carcinogenicity|No human data and inadequate animal data.|0490
Bromoform|75-25-2|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Based on inadequate human data and sufficient evidence of carcinogenicity in animals, namely an increased incidence of tumors after oral administration of bromoform in rats and intraperitoneal administration in mice. Bromoform is genotoxic in several assay systems. Also, bromoform is structurally related to other trihalomethanes (e.g., chloroform, bromodichloromethane, dibromochloromethane) which have been verified as either probable or possible carcinogens.|0214
Bromomethane|74-83-9|D, Not classifiable as to human carcinogenicity|Inadequate human and animal data: a single mortality study from which direct exposure associations could not be deduced and studies in several animal species with too few animals, too brief exposure or observation time for adequate power. Bromomethane has shown genotoxicity.|0015
Bromotrichloromethane|75-62-7|D, Not classifiable as to human carcinogenicity|Based on no data regarding the carcinogenicity of bromotrichloromethane in humans or animals.|0533
Bromoxynil|1689-84-5|Not Assessed under the IRIS program.||0289
Bromoxynil octanoate|1689-99-2|Not Assessed under the IRIS program.||0138
1,3-Butadiene|106-99-0|NA, Not applicable. This substance was not assessed using the 1986 cancer guidelines (U.S. EPA, 1986).|Under EPA's 1999 Guidelines for Carcinogen Risk Assessment (U.S. EPA, 1999), 1,3-butadiene is characterized as carcinogenic to humans by inhalation. This characterization is supported by the total weight of evidence provided by the following: (1) sufficient evidence from epidemiologic studies of the majority of U.S. workers occupationally exposed to 1,3-butadiene, either to the monomer or to the polymer by inhalation, showing increased lymphohematopoietic cancers and a dose-response relationship for leukemias in polymer workers (see Section II.A.2), (2) sufficient evidence in laboratory animal studies showing that 1,3-butadiene causes tumors at multiple sites in mice and rats by inhalation (see Section II.A.3), and (3) numerous studies consistently demonstrating that 1,3-butadiene is metabolized into genotoxic metabolites by experimental animals and humans (see Section II.A.4). The specific mechanisms of 1,3-butadiene-induced carcinogenesis are unknown; however, the scientific evidence strongly suggests that the carcinogenic effects are mediated by genotoxic metabolites of 1,3-butadiene, i.e., the monoepoxide, the diepoxide, and the epoxydiol.|0139
n-Butanol|71-36-3|D, Not classifiable as to human carcinogenicity|Based on no human and no animal cancer data.|0140
Butyl benzyl phthalate|85-68-7|C, Possible human carcinogen|Based on statistically significant increase in mononuclear cell leukemia in female rats; the response in male rats was inconclusive and there was no such response in mice.|0293
Butylate|2008-41-5|Not Assessed under the IRIS program.||0215
t-Butylchloride|507-20-0|D, Not classifiable as to human carcinogenicity|Based on no human carcinogenicity data and inadequate animal data.|0417
Butylphthalyl butylglycolate (BPBG)|85-70-1|Not Assessed under the IRIS program.||0016
Cacodylic acid|75-60-5|D, Not classifiable as to human carcinogenicity|No human data and inadequate data in animals|0587
Cadmium|7440-43-9|B1, Probable human carcinogen - based on limited evidence of carcinogenicity in humans|Limited evidence from occupational epidemiologic studies of cadmium is consistent across investigators and study populations. There is sufficient evidence of carcinogenicity in rats and mice by inhalation and intramuscular and subcutaneous injection. Seven studies in rats and mice wherein cadmium salts (acetate, sulfate, chloride) were administered orally have shown no evidence of carcinogenic response.|0141
Calcium cyanide|592-01-8|Not Assessed under the IRIS program.||0017
Caprolactam|105-60-2|Not Assessed under the IRIS program.||0357
Captafol|2425-06-1|Not Assessed under the IRIS program.||0216
Captan|133-06-2|Not Assessed under the IRIS program.||0018
Carbaryl|63-25-2|Not Assessed under the IRIS program.||0019
Carbofuran|1563-66-2|Not Assessed under the IRIS program.||0218
Carbon disulfide|75-15-0|Not Assessed under the IRIS program.||0217
Carbon tetrachloride|56-23-5|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Carcinogenicity in rats, mice, and hamsters|0020
Carbonyl sulfide|463-58-1|Not Assessed under the IRIS program.||0617
Carbosulfan|55285-14-8|Not Assessed under the IRIS program.||0021
Carboxin|5234-68-4|Not Assessed under the IRIS program.||0022
Cerium Oxide and Cerium Compounds|1306-38-3|||1018
Chloral hydrate|302-17-0|C, Possible human carcinogen|Under the 1996 proposed guidelines for carcinogen risk assessment (U.S. EPA, 1996), chloral hydrate shows suggestive evidence of human carcinogenicity by the oral route of exposure.|0304
Chloramben|133-90-4|Not Assessed under the IRIS program.||0023
Chlordane (Technical)|12789-03-6|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Under the 1996 Proposed Guidelines, chlordane would be characterized as a likely carcinogen by all routes of exposure.|0142
Chlordecone (Kepone)|143-50-0|||1017
Chlorimuron-ethyl|90982-32-4|Not Assessed under the IRIS program.||0406
Chlorine|7782-50-5|Not Assessed under the IRIS program.||0405
Chlorine cyanide|506-77-4|Not Assessed under the IRIS program.||0024
Chlorine dioxide|10049-04-4|D, Not classifiable as to human carcinogenicity|Under the draft Carcinogen Assessment Guidelines (U.S. EPA, 1996), the human carcinogenicity of chlorine dioxide cannot be determined because no satisfactory human or animal studies assessing the chronic carcinogenic potential of chlorine dioxide have been located.|0496
Chlorite (sodium salt)|7758-19-2|D, Not classifiable as to human carcinogenicity|Under the draft Carcinogen Assessment Guidelines (U.S. EPA, 1996), the human carcinogenicity of chlorite cannot be determined because of a lack of human data and limitations in animal studies.|0648
1-Chloro-1,1-difluoroethane|75-68-3|Not Assessed under the IRIS program.||0661
2-Chloroacetophenone|532-27-4|Not Assessed under the IRIS program.||0537
p-Chloroaniline|106-47-8|Not Assessed under the IRIS program.||0320
Chlorobenzene|108-90-7|D, Not classifiable as to human carcinogenicity|No human data, inadequate animal data and predominantly negative genetic toxicity data in bacterial, yeast, and mouse lymphoma cells.|0399
Chlorobenzilate|510-15-6|Not Assessed under the IRIS program.||0400
1-Chlorobutane|109-69-3|D, Not classifiable as to human carcinogenicity|Based on no human carcinogenicity data and inadequate animal data.|0415
2-Chlorobutane|78-86-4|D, Not classifiable as to human carcinogenicity|Based on no human carcinogenicity data and inadequate animal data.|0416
Chlorocyclopentadiene|41851-50-7|D, Not classifiable as to human carcinogenicity|Lack of data concerning carcinogenicity in humans or animals.|0430
Chlorodifluoromethane|75-45-6|Not Assessed under the IRIS program.||0657
Chloroform|67-66-3|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|Under the Proposed Guidelines for Carcinogen Risk Assessment (U.S. EPA, 1996; U.S. EPA, 1999), chloroform is likely to be carcinogenic to humans by all routes of exposure under high-exposure conditions that lead to cytotoxicity and regenerative hyperplasia in susceptible tissues (U.S. EPA, 1998a,b). Chloroform is not likely to be carcinogenic to humans by any route of exposure under exposure conditions that do not cause cytotoxicity and cell regeneration. This weight-of-evidence conclusion is based on: 1) observations in animals exposed by both oral and inhalation pathways which indicate that sustained or repeated cytotoxicity with secondary regenerative hyperplasia precedes, and is probably required for, hepatic and renal neoplasia; 2) there are no epidemiological data specific to chloroform and, at most, equivocal epidemiological data related to drinking water exposures that cannot necessarily be negative, although there are some scattered positive results that generally have limitations such as excessively high dose or with confounding factors. Thus, the weigh-of-evidence of the genotoxicity data on chloroform supports a conclusion that chloroform is not strongly mutagenic, and the genotoxicity is not likely to be the predominant mode of action underlying the carcinogenic potential of chloroform. Although no cancer data exist for exposures via the dermal pathway, the weight-of-evidence conclusion is considered to be applicable to this pathway as well, because chloroform absorbed through the skin and into the blood is expected to be metabolized and to cause toxicity in much the same way as chloroform absorbed by other exposure routes.|0025
Chloromethyl methyl ether (CMME)|107-30-2|A, Human Carcinogen|The observation of an increased incidence of respiratory cancer in exposed workers and the observation of respiratory tumors in mice, rats, and hamsters exposed by inhalation forms the basis for this classification.|0245
beta-Chloronaphthalene|91-58-7|Not Assessed under the IRIS program.||0463
2-Chlorophenol|95-57-8|Not Assessed under the IRIS program.||0303
p-Chlorophenyl methyl sulfide|123-09-1|D, Not classifiable as to human carcinogenicity|No human or animal studies found in the available literature|0623
p-Chlorophenyl methyl sulfone|98-57-7|D, Not classifiable as to human carcinogenicity|No human or animal studies found in the available literature|0624
p-Chlorophenyl methyl sulfoxide|934-73-6|D, Not classifiable as to human carcinogenicity|No human or animal studies found in the available literature|0625
Chloroprene|126-99-8|||1021
Chlorothalonil|1897-45-6|Not Assessed under the IRIS program.||0143
o-Chlorotoluene|95-49-8|Not Assessed under the IRIS program.||0412
Chlorpropham|101-21-3|Not Assessed under the IRIS program.||0283
Chlorpyrifos|2921-88-2|Not Assessed under the IRIS program.||0026
Chlorsulfuron|64902-72-3|Not Assessed under the IRIS program.||0027
Chromium(III), insoluble salts|16065-83-1|D, Not classifiable as to human carcinogenicity|Using the Proposed Guidelines for Carcinogen Risk Assessment (EPA, 1996), there are inadequate data to determine the potential carcinogenicity of trivalent chromium, as discussed below. However, the classification of hexavalent chromium as a known human carcinogen raises a concern for the carcinogenic potential of trivalent chromium.|0028
Chromium(VI)|18540-29-9|A, Human Carcinogen (Inhalation route)D, Not classifiable as to human carcinogenicity (Oral route)|Under the proposed guidelines (EPA, 1996), Cr(VI) would be characterized as a known human carcinogen by the inhalation route of exposure. The oral carcinogenicity of Cr(VI) cannot be determined. No data were located in the available literature that suggested that Cr(VI) is carcinogenic by the oral route of exposure.|0144
Chrysene|218-01-9|B2, Probable human carcinogen - based on sufficient evidence of carcinogenicity in animals|No human data and sufficient data from animal bioassays. Chrysene produced carcinomas and malignant lymphoma in mice after intraperitoneal injection and skin carcinomas in mice following dermal exposure. Chrysene produced chromosomal abnormalities in hamsters and mouse germ cells after gavage exposure, positive responses in bacterial gene mutation assays and transformed mammalian cells exposed in culture.|0455
Coke oven emissions|8007-45-2|A, Human Carcinogen|Studies of coke oven workers have shown increased risk of mortality from cancer of the lung, trachea and bronchus; cancer of the kidney; cancer of the prostate; and cancer at all sites combined. In animals, extracts and condensates of coke oven emissions were found to be carcinogenic in both inhalation studies and skin-painting bioassays. The mutagenicity of whole extracts and condensates, as well as their individual components, provides supportive evidence for carcinogenicity.|0395
Copper|7440-50-8|D, Not classifiable as to human carcinogenicity|There are no human data, inadequate animal data from assays of copper compounds, and equivocal mutagenicity data.|0368
Copper cyanide|544-92-3|Not Assessed under the IRIS program.||0029
Creosote|8001-58-9|B1, Probable human carcinogen - based on limited evidence of carcinogenicity in humans|Limited evidence of the association between occupational creosote contact and subsequent tumor formation, sufficient evidence of local and distant tumor formation after dermal application to mice, and some evidence of mutagenic activity, as well as the well-documented carcinogenicity of other coal tar products to humans.|0360
Crotonaldehyde|123-73-9|C, Possible human carcinogen|Based on no human data and an increased incidence of hepatocellular carcinomas and hepatic neoplastic nodules (combined) in male F344 rats. The possible carcinogenicity of crotonaldehyde is supported by genotoxic activity and the expected reactivity of croton oil and aldehyde. Crotonaldehyde is also a suspected metabolite of N-nitrosopyrrolidine, a probable human carcinogen.
</t2b>

Latest revision as of 19:33, 17 May 2014

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<display_map type="terrain"> 62.904602, 27.647781 62.891918, 27.680013 </display_map>


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Sandbox: Difference between revisions(kg)
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